Download e-book for iPad: Body MRI by Evan Siegelman

By Evan Siegelman

ISBN-10: 072163740X

ISBN-13: 9780721637402

This functional new reference makes it effortless to judge ailment methods of the chest, stomach, and pelvis. greater than 1,000 MR photographs illustrate common anatomy and the entities readers in most cases stumble upon in perform. a number of key issues bins in every one bankruptcy make medical assistance effortless to discover and follow. It additionally addresses the increasing use of MRI to ascertain the female and male reproductive platforms, pelvis, hips, bladder, and breast.

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Extra resources for Body MRI

Sample text

1-17; see Fig. 29Hepatic metastases with In patients with breast cancer referred to high protein synthesis may be present in multiple myeloma and c a r ~ i n o i dMucin . ^^^,^^^ s t m ~ t u r e s The . 291 2391315,316 - Chap Liver . Figure 1-16 MR demonstration of wesectable metastatic breast cancer. A, Fat-suppressed T2-WI shows multiple variably sized hepatic masses. The metastases have a peripheral rim (arrows) that is hyperintense to liver parenchyma and nearly isointense to spleen. The central portions of some of the masses are hyperintense (*) to the tumor periphery owing to the presence of necrosis or immature fibrosis.

404,405 Thrombus less than 5 weeks old appears hyperintense to liver parenchyma on T1- and T2-WIs, whereas thrombus between 2 and 18 months old appears hyperintense only on T ~ - W I S . ~Potential O~ pitfalls on unenhanced TI- and T2-WI include slow or turbulent . Figure 1-25 MR findings of cavernous transformation of the portal vein secondary to extrinsic compression by a (previously excised) ciliated hepatic foregut cyst. Axial maximum intensity projection (MIP) image from portal venous phase of a CE TI-WI shows replacement of the intrahepatic portal vein by numerous small collateral vessels (arrow).

D, T2*-W GRE image (TR = 100, TE = 20, FA = 20") reveals markedly hypointense liver (L). The pancreas (P) does not lose SI to indicate parenchymal iron deposition. The spleen (S) is not involved with the parenchymal iron deposition of genetic hemochromatosis. The low SI within the bone marrow is not secondary to iron deposition. Instead, susceptibility effects of bony trabeculae are responsible for the T2* effects. 379 Hypercoagulable states Patients with cirrhosis unrelated to GH can develop diffuse hepatic iron d e p o s i t i ~ n .

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Body MRI by Evan Siegelman

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