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By Adler R.

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M. Buccoliero et al. extra copy of some genes and is missing copies of other genes. i17p correlates with histological variants and predicts the survival. i17q is more frequently observed in classic and anaplastic/ large cell medulloblastoma and has been associated with a poor clinical outcome when compared with that of desmoplastic/nodular medulloblastoma and medulloblastoma with extensive nodularity. This observation suggests that this cytogenetic alteration may contribute to the development of aggressive histotypes (Lamont et al.

Deregulated expression of MYCs is often associated with aggressive, poorly differentiated tumors. High-level amplification (>10-fold) of MYC oncogenes is particularly associated with the large cell/anaplastic phenotype. A moderate MYC oncogenes amplification (>5-fold but <10fold), particularly c-MYC and N-MYC, is sometimes detected in other histological subtypes and may depend on the presence a large cell/anaplastic component in this medulloblastomas (Eberhart et al. 2004; Stearns et al. 2006; Takei et al.

1999). Treatment Options for Aggressive Oligodendroglial Tumors Aggressive and recurrent oligodendroglial tumors are challenging to treat. Although extensive surgical resection has been shown, in two prospective studies, to be associated with increased survival (Cairncross et al. 2006; van den Bent et al. 2006), further adjuvant treatment (radiation or chemotherapy) is required in patients with these highergrade tumors to prevent recurrence (van den Bent 2007). Since oligodendroglial tumors were found to be sensitive to PCV chemotherapy in the mid 1990s (Cairncross et al.

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An Intro to Continuity, Extrema & Related Topics for general gaussian process by Adler R.

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